Wei L, Cafiero T, Tseng A, Gertje H, Berneshawi A, Crossland N, Ploss A. Conversion of hepatitis B virus relaxed circular to covalently closed circular DNA is supported in murine cells. JHEP Rep. 2022;4(9):100534. PMCID: PMC9403495

Biswas S, Rust L, Wettengel J, Yusova S, Fischer M, Carson J, Johnson J, Wei L, Thode T, Kaadige M, Sharma S, Agbaria M, Bimber B, Tu T, Protzer U, Ploss A, Smedley J, Golomb G, Sacha J, Burwitz B. Long-term hepatitis B virus infection of rhesus macaques requires suppression of host immunity. Nat Commun. 2022;13(1):2995. PMCID: PMC9151762

Wei L, Ploss A. Rise above the stress-Endoplasmic reticulum stress and autophagy enhance the release of hepatitis B virus subparticles. Hepatology. 2022;75(2):248–251. PMID: 34890054

Wei L, Ploss A. Mechanism of Hepatitis B Virus cccDNA Formation. Viruses. 2021;13(8). PMCID: PMC8402782

Wei L, Ploss A. Hepatitis B virus cccDNA is formed through distinct repair processes of each strand. Nat Commun. 2021;12(1):1591. PMCID: PMC7952586

Wei L, Ploss A. Core components of DNA lagging strand synthesis machinery are essential for hepatitis B virus cccDNA formation. Nat Microbiol. 2020;5(5):715–726. PMCID: PMC7190442

Ding Q, Gaska J, Douam F, Wei L, Kim D, Balev M, Heller B, Ploss A. Species-specific disruption of STING-dependent antiviral cellular defenses by the Zika virus NS2B3 protease. Proc Natl Acad Sci U S A. 2018;115(27):E6310-E6318. PMCID: PMC6142274